这是一篇来自已证抗体库的有关人类 Kir4.1 (Kir4.1) 的综述，是根据10篇发表使用所有方法的文章归纳的。这综述旨在帮助来邦网的访客找到最适合Kir4.1 抗体。
Kir4.1 同义词: BIRK-10; KCNJ13-PEN; KIR1.2; KIR4.1; SESAME; ATP-sensitive inward rectifier potassium channel 10; ATP-dependent inwardly rectifying potassium channel Kir4.1; glial ATP-dependent inwardly rectifying potassium channel KIR4.1; inward rectifier K(+) channel Kir1.2; inward rectifier K+ channel KIR1.2; potassium channel, inwardly rectifying subfamily J member 10; potassium inwardly-rectifying channel, subfamily J, member 10

Alomone Labs
豚鼠 多克隆
  • 免疫组化-冰冻切片; 大鼠; 1:200; 图 6c
Alomone LabsKir4.1抗体（Alomone Labs, AGP-012)被用于被用于免疫组化-冰冻切片在大鼠样品上浓度为1:200 (图 6c). J Comp Neurol (2019) ncbi
兔 多克隆
  • 免疫组化; 小鼠; 1:300; 图 5
Alomone LabsKir4.1抗体（Alomone, APC-035)被用于被用于免疫组化在小鼠样品上浓度为1:300 (图 5). Sci Rep (2016) ncbi
兔 多克隆
  • 免疫组化-石蜡切片; 人类; 4 ug/ml; 图 3
Alomone LabsKir4.1抗体（Alomone, APC-035)被用于被用于免疫组化-石蜡切片在人类样品上浓度为4 ug/ml (图 3). Acta Neuropathol Commun (2016) ncbi
兔 多克隆
  • 免疫组化-冰冻切片; 人类; 1:500; 图 7
Alomone LabsKir4.1抗体（Alomone Labs, APC-035)被用于被用于免疫组化-冰冻切片在人类样品上浓度为1:500 (图 7). Cell Tissue Res (2016) ncbi
兔 多克隆
  • 免疫组化-冰冻切片; 大鼠; 1:600; 图 4a
  • 免疫印迹; 大鼠; 1:800; 图 2a
Alomone LabsKir4.1抗体（Alomone Labs, APC-035)被用于被用于免疫组化-冰冻切片在大鼠样品上浓度为1:600 (图 4a) 和 被用于免疫印迹在大鼠样品上浓度为1:800 (图 2a). Neuroscience (2016) ncbi
兔 多克隆
  • 免疫组化-石蜡切片; 人类; 图 1
Alomone LabsKir4.1抗体（Alomone Labs, APC-035)被用于被用于免疫组化-石蜡切片在人类样品上 (图 1). Ann Neurol (2014) ncbi
兔 多克隆
  • 免疫组化-石蜡切片; 小鼠
Alomone LabsKir4.1抗体（Alomone, APC-035)被用于被用于免疫组化-石蜡切片在小鼠样品上. PLoS ONE (2013) ncbi
兔 多克隆
  • 免疫印迹; 小鼠; 1:1000
Alomone LabsKir4.1抗体（Alomone, APC-035)被用于被用于免疫印迹在小鼠样品上浓度为1:1000. Am J Physiol Renal Physiol (2008) ncbi
武汉三鹰
兔 多克隆
武汉三鹰Kir4.1抗体（Proteintech, 12503-1-AP)被用于. Glia (2015) ncbi
默克密理博中国
兔 多克隆
  • 免疫组化; 小鼠; 1:200
默克密理博中国Kir4.1抗体（Chemicon, AB5818)被用于被用于免疫组化在小鼠样品上浓度为1:200. FASEB J (2015) ncbi
兔 多克隆
  • 免疫组化; 小鼠; 1:200
默克密理博中国Kir4.1抗体（Chemicon, AB5818)被用于被用于免疫组化在小鼠样品上浓度为1:200. FASEB J (2015) ncbi
西格玛奥德里奇
小鼠 单克隆（1C11）
  • 免疫组化-石蜡切片; 人类; 1:100
西格玛奥德里奇Kir4.1抗体（Sigma-Aldrich, WH0003766M1)被用于被用于免疫组化-石蜡切片在人类样品上浓度为1:100. Ann Neurol (2014) ncbi
文章列表
  1. Koike T, Tanaka S, Hirahara Y, Oe S, Kurokawa K, Maeda M, et al. Morphological characteristics of p75 neurotrophin receptor-positive cells define a new type of glial cell in the rat dorsal root ganglia. J Comp Neurol. 2019;: pubmed 出版商
  2. Ingham N, Carlisle F, Pearson S, Lewis M, Buniello A, Chen J, et al. S1PR2 variants associated with auditory function in humans and endocochlear potential decline in mouse. Sci Rep. 2016;6:28964 pubmed 出版商
  3. Koeppen A, Ramirez R, Becker A, Mazurkiewicz J. Dorsal root ganglia in Friedreich ataxia: satellite cell proliferation and inflammation. Acta Neuropathol Commun. 2016;4:46 pubmed 出版商
  4. Liu W, Edin F, Blom H, Magnusson P, Schrott Fischer A, Glueckert R, et al. Super-resolution structured illumination fluorescence microscopy of the lateral wall of the cochlea: the Connexin26/30 proteins are separately expressed in man. Cell Tissue Res. 2016;365:13-27 pubmed 出版商
  5. Najafi E, Stoodley M, Bilston L, Hemley S. Inwardly rectifying potassium channel 4.1 expression in post-traumatic syringomyelia. Neuroscience. 2016;317:23-35 pubmed 出版商
  6. Minkel H, Anwer T, Arps K, Brenner M, Olsen M. Elevated GFAP induces astrocyte dysfunction in caudal brain regions: A potential mechanism for hindbrain involved symptoms in type II Alexander disease. Glia. 2015;63:2285-97 pubmed 出版商
  7. Martínez Vega R, Garrido F, Partearroyo T, Cediel R, Zeisel S, Martínez Álvarez C, et al. Folic acid deficiency induces premature hearing loss through mechanisms involving cochlear oxidative stress and impairment of homocysteine metabolism. FASEB J. 2015;29:418-32 pubmed 出版商
  8. Schirmer L, Srivastava R, Kalluri S, Böttinger S, Herwerth M, Carassiti D, et al. Differential loss of KIR4.1 immunoreactivity in multiple sclerosis lesions. Ann Neurol. 2014;75:810-28 pubmed 出版商
  9. Buniello A, Hardisty Hughes R, Pass J, Bober E, Smith R, Steel K. Headbobber: a combined morphogenetic and cochleosaccular mouse model to study 10qter deletions in human deafness. PLoS ONE. 2013;8:e56274 pubmed 出版商
  10. Singh R, Wangemann P. Free radical stress-mediated loss of Kcnj10 protein expression in stria vascularis contributes to deafness in Pendred syndrome mouse model. Am J Physiol Renal Physiol. 2008;294:F139-48 pubmed