这是一篇来自已证抗体库的有关人类 TERF2IP的综述,是根据6篇发表使用所有方法的文章归纳的。这综述旨在帮助来邦网的访客找到最适合TERF2IP 抗体。
TERF2IP 同义词: DRIP5; RAP1; telomeric repeat-binding factor 2-interacting protein 1; TERF2-interacting telomeric protein 1; TRF2-interacting telomeric RAP1 protein; dopamine receptor-interacting protein 5; repressor/activator protein 1 homolog

Bethyl
兔 多克隆
  • 免疫印迹; 人类; 1:2000; 图 s6
Bethyl TERF2IP抗体(Bethyl, A300-306A)被用于被用于免疫印迹在人类样品上浓度为1:2000 (图 s6). PLoS ONE (2018) ncbi
兔 多克隆
  • 免疫印迹; 人类; 1:500; 图 S1C
Bethyl TERF2IP抗体(Bethyl, A300-306A)被用于被用于免疫印迹在人类样品上浓度为1:500 (图 S1C). J Cell Sci (2017) ncbi
兔 多克隆
  • 免疫印迹; 人类; 1:2000; 图 3
Bethyl TERF2IP抗体(Bethyl Laboratories, A300-306A)被用于被用于免疫印迹在人类样品上浓度为1:2000 (图 3). Biomed Rep (2016) ncbi
兔 多克隆
  • 免疫印迹; 人类; 图 4d
Bethyl TERF2IP抗体(Bethyl Laboratory, A300-306A)被用于被用于免疫印迹在人类样品上 (图 4d). J Cell Sci (2016) ncbi
兔 多克隆
  • 免疫印迹; 人类; 图 4
Bethyl TERF2IP抗体(Bethyl Laboratories, A300-306A)被用于被用于免疫印迹在人类样品上 (图 4). Aging Cell (2015) ncbi
西格玛奥德里奇
小鼠 单克隆(4c8/1)
  • 免疫沉淀; 人类; 10 ug
  • 免疫印迹; 人类
西格玛奥德里奇 TERF2IP抗体(Sigma, 4C8/1)被用于被用于免疫沉淀在人类样品上浓度为10 ug 和 被用于免疫印迹在人类样品上. EMBO J (2010) ncbi
文章列表
  1. Brosnan Cashman J, Yuan M, Graham M, Rizzo A, Myers K, Davis C, et al. ATRX loss induces multiple hallmarks of the alternative lengthening of telomeres (ALT) phenotype in human glioma cell lines in a cell line-specific manner. PLoS ONE. 2018;13:e0204159 pubmed 出版商
  2. Zanini I, Soneson C, Lorenzi L, Azzalin C. Human cactin interacts with DHX8 and SRRM2 to assure efficient pre-mRNA splicing and sister chromatid cohesion. J Cell Sci. 2017;130:767-778 pubmed 出版商
  3. Swanson M, Baribault M, Israel J, Bae N. Telomere protein RAP1 levels are affected by cellular aging and oxidative stress. Biomed Rep. 2016;5:181-187 pubmed
  4. Hong J, Lee J, Chung I. Telomerase activates transcription of cyclin D1 gene through an interaction with NOL1. J Cell Sci. 2016;129:1566-79 pubmed 出版商
  5. Luo Z, Feng X, Wang H, Xu W, Zhao Y, Ma W, et al. Mir-23a induces telomere dysfunction and cellular senescence by inhibiting TRF2 expression. Aging Cell. 2015;14:391-9 pubmed 出版商
  6. Bombarde O, Boby C, Gomez D, Frit P, Giraud Panis M, Gilson E, et al. TRF2/RAP1 and DNA-PK mediate a double protection against joining at telomeric ends. EMBO J. 2010;29:1573-84 pubmed 出版商