这是一篇有关其他小鼠品系或模型的综述,是根据62篇发表使用实验的文章归纳的。这综述旨在帮助来邦网的访客找到最适合其他小鼠品系或模型。
赛默飞世尔
为了研究神经调节蛋白(NRG)和BDNF对少突胶质细胞的NMDA受体依赖性髓鞘形成过程的影响,采用了Molecular Probes公司的羊抗小鼠二抗,进行免疫细胞化学实验。至该文献
Jackson ImmunoResearch Laboratories
为了研究再循环的视紫红质在保持光受体蛋白功能和完整性过程中Retromer复合物的作用,采用了Jackson ImmunoResearch公司的抗鼠二抗,进行免疫组化实验。至该文献
丹科医疗器械技术服务(上海)有限公司
为了研究神经调节蛋白(NRG)和BDNF对少突胶质细胞的NMDA受体依赖性髓鞘形成过程的影响,采用了Dako公司的抗小鼠二抗,进行免疫印迹实验。至该文献
Charles River Laboratories
为了研究通过运动或基因操作引起的Capicua表达降低能够改善小脑脊髓运动失调1型的症状,采用Charles River的E15.5 mouse embryo进行神经元培养实验。至该文献
为了研究抗氧化反应能够通过抑制丙酮酸激酶M2的活性来实现,采用Charles River的nu/nu mice进行异种移植实验。至该文献
为了研究来自精子的线粒体和其他细胞器在受精后会被清除,采用Charles River的female OF1 mouse进行超排卵实验。至该文献
为了研究小鼠血糖能够通过人工合成的光遗传学转录元件进行调控,采用Charles River Laboratory的female OF1 mice进行腹腔移植实验。至该文献
为了研究alpha2A肾上腺素受体与2型糖尿病之间的联系,使用了Charles River Laboratories公司的F344/DuCrl2Swe小鼠来作为模式生物。至该文献
Jackson Laboratories显示:30; 总数:43
为了研究巨噬细胞对成体毛囊干细胞的调控,采用了Jackson Lab公司的K15-GFP模型小鼠,进行种间杂交实验。至该文献
为了研究哺乳动物嗅觉系统,采用了Jackson Laboratory公司的GCAMP3报告系统转基因小鼠,进行种间杂交实验。至该文献
为了研究NDE1蛋白在早期神经祖细胞分化的S期的功能,采用了JaxMice公司的Trp53基因敲除小鼠,进行种间杂交实验。至该文献
为了研究小鼠中背侧纹状体的DR1神经元对食物预期的昼夜活动节律的调控,采用了Jackson laboratory公司的D2R knockout mice(D2R敲除小鼠),进行种间杂交实验。至该文献
为了研究mTORC1在红细胞生成和贫血症中的作用,采用了Jackson laboratory公司的Vav-iCre mice(Vav-iCre小鼠),进行种间杂交实验。至该文献
为了研究抗肿瘤反应中Dectin-1的作用,采用了Jackson Laboratory公司的CD11c-DTR小鼠,进行种间杂交实验。至该文献
为研究新皮层第五层锥体神经元的时间编码信息受到近胞体抑制的特别活化的调控,采用Jackson Laboratory的SST-cre mice进行电生理实验。至该文献
为了研究炎症过程中造血系统与多种器官(包括脑部)之间基于RNA的信号传递机制,采用了JAX的Rosa26-LacZ小鼠模型,进行杂交实验。至该文献
为了研究I型干扰素在多聚IC处理的树突细胞成熟过程中的作用,采用了Jackson Laboratory公司的Hif1-alpha-flox/flox小鼠,进行杂交实验。至该文献
为了研究成年哺乳动物体内,刺激信号传导对外周神经系统中线粒体转运的影响,采用了Jackson laboratories公司的Thy-1-YFP-16 positive or negative littermates动物模型,进行杂交实验。至该文献
为了研究胞质凝聚过程在亨廷顿病的分子病理中的作用,采用了Jackson Laboratory公司的CHDI colonies,进行种间杂交实验。至该文献
为了研究B细胞受体信号通路调控过程中N-wasp的作用,采用了Jackson Laboratories 公司的野生CBA/CaJ鼠,进行种间杂交实验。至该文献
为了在小鼠模型中研究sFlt-1对于维持无血管感光层的作用,The Jackson Laboratory的Gt(ROSA)26Sortm4(ACTB-tdTomato,-EGFP)Luo/J小鼠被用于进行动物实验。至该文献
为了研究小鼠大脑中胆固醇传感器SCAP降低引起的突触传导和认知能力损伤,采用了The Jackson Laboratory的db/db mice (C57Bl/Ks background)获得转基因小鼠。至该文献
为了研究MAPK异常活化可被Ste5构象改变隔绝,采用了Jackson Laboratory的Bmal 1 +/- mice进行小鼠杂交实验。至该文献
为了研究母婴免疫耐受被蜕膜基质细胞中的趋化因子基因失活调控,采用了The Jackson Laboratory的hemizygous Act-mOVA males进行生化实验。至该文献
为了研究胰岛素抵抗能够被雷帕霉素通过mTORC2 受损诱导,采用了Jackson Laboratory的Ubiquitin C-CreERT2 mice进行生化实验。至该文献
为了研究特定的骨髓细胞源自卵黄囊,采用了The Jackson Laboratory的Rosa26LSL-YFP reporter mice进行生化实验。至该文献
为了研究朗格汉斯细胞介导的PAH代谢能导致DNA损伤和肿瘤发生,采用The Jackson Laboratory的FVB/N mice进行肿瘤诱导实验。至该文献
为了研究生发中心的B细胞会进行不对称分裂,采用Jackson Laboratories的Icam1-/- mice进行生物化学实验。至该文献
为了研究小肠淋巴滤泡的形成需要食物中的配体来激活AhR,采用Jackson Laboratories的Itgax-CreTg mice进行生化实验。至该文献
为了研究小肠中宿主与细菌的共生关系受凝集素RegIIIgamma保护免受免疫系统攻击,采用了Jackson Laboratories的Villin-Cre mice进行条件性敲除小鼠制备实验。至该文献
为了研究迷走神经环路通过调控分泌乙酰胆碱的T细胞来抑制脾脏中细胞因子的分泌,采用The
Jackson Laboratory的ChAT(BAC)-eGFP小鼠进行生化实验。至该文献
为了研究氧化应激条件下DNA修复能被SIRT6通过PARP1活化加强,采用Jackson Lab的SIRT6-/+ heterozygous mice进行细胞培养实验。至该文献
为了研究溶酶体形成能够被TFEB调控并导致细胞自噬,采用Jackson laboratory的Albumin-CRE mice进行小鼠模型建立实验。至该文献
为了研究脆弱拟杆菌(Bacteroides fragilis)在宿主-微生物共生的建立中的作用,采用Jackson Labs的Tlr2-/-小鼠提取其细胞。至该文献
为了揭示CTLA-4为何能够抑制CD28的激活,采用Jackson Laboratory的DO11.10 TCR转基因小鼠提取相应细胞。至该文献
为了阐述沉默朊蛋白可以加速通过T细胞信号的中枢神经系统的自我免疫,采用了杰克逊实验室的OVA323-339 TCR 转基因 (OT2)小鼠为模型进行了试验。至该文献
为了研究KLF家族成员对中枢神经系统神经元再生能力的调节,使用了Jackson Laboratories公司的N-Tg(Thy1-cre)1Vln/J 小鼠作为模式生物。至该文献
为证实p53的缺失会导致缺乏p21的间质干细胞的肿瘤发生,使用了Jackson Laboratory公司的Cg-Prkdcscid IL2rgtm1Wjl/SzJ小鼠来进行研究。至该文献
Taconic
为了研究PICK1缺乏引起的分泌小泡生成损伤、生长阻滞及乳糖耐受降低,采用了Taconic Farms, Inc.公司的对照瘦鼠进行小鼠基因学实验至该文献
为了研究针对共生菌的免疫反应会被急性胃肠炎诱导,采用了Taconic的OT-II TCR transgenic mice进行寄生虫感染实验。至该文献
为了研究皮肤微生物能够提供保护性免疫,采用了Taconic Farms的Il-1r1-/- mice进行动物实验。至该文献
为了研究母婴免疫耐受被蜕膜基质细胞中的趋化因子基因失活调控,采用了Taconic Farms的vasectomized ICR males进行生化实验。至该文献
为了研究NKT细胞能被早期接触到的微生物调控,采用了Taconic USA的Swiss Webster (SW) mice进行生物化学实验。至该文献
为了揭示CTLA-4为何能够抑制CD28的激活,采用Taconic Farms的RAG-2-/-小鼠提取相应细胞。至该文献
为了研究风湿性关节炎小鼠模型能够利用progranulin通过与TNF受体的结合加以治疗,采用Taconic的TNFalpha转基因小鼠进行风湿性关节炎治疗实验。至该文献
为确定二肽基肽酶IV在免疫系统的作用,实验中应用了Taconic Laboratory提供的DPP-IV-/-小鼠。至该文献
为了阐明在自然杀伤T淋巴细胞发育过程中,钙调磷酸酶-NFAT-Egr2 途径的重要作用,采用了Taconic Farms的Egr1−/− 小鼠作为模式生物。至该文献
为了说明Rcan1 对FcεRI调节的信号途径和肥大细胞功能所起的负调节作用,采用了Taconic的Egr1缺陷型小鼠作为模式生物。至该文献
为证实淋巴组织诱导物类似细胞可以产生白介素17和白介素22,使用了Taconic 的Rag2 − / − 小鼠来作为模式生物。至该文献
为说明CD4+T细胞介导的针对HSV-2的免疫应答反应会由于IL-15的过量表达而降低,使用了Taconic公司的RAG1−/−小鼠来作为模式动物。至该文献
Individual Researcher
为了研究一些65KD的G蛋白在保护宿主免受细菌感染中的作用,采用Texas基因组医学研究院的Gbp1基因敲除小鼠用于组织提取。至该文献
文章列表
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